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OTOFEarly clinicalActive

OTOV101N + OTOV101C

Study sponsor: Otovia Therapeutics

An investigator-initiated program evaluating a dual-vector OTOF gene-replacement approach in people with DFNB9 hearing loss.

This is an investigational therapy. Inclusion here does not mean it is approved, available, safe, or effective.

Plain-language summary

Study overview

Sources
Current study status
Active
Who is being studied
1 year and older
How treatment is given
Intracochlear administration
Study start date
Jun 26, 2023

Study start date is reported by ClinicalTrials.gov and may differ from the date the first participant received treatment.

Official study record

NCT05901480 · Status shown: Active · Registry information last checked not recently verified

Terminology Dictionary
Target gene
The gene the treatment is designed to address.
Gene replacement
Delivery of a working gene copy intended to restore a missing or impaired function.
Vector / payload
The delivery vehicle and the genetic material it carries.
Promoter
A DNA control sequence that helps determine where and how strongly the delivered gene is active.
Cochlear access
The route used to reach the inner ear and administer the treatment.
Laterality
Whether treatment is given to one ear or both ears.
Phase 1 / 2
An early human study designed mainly to examine safety, dosing, and initial signs of biological or clinical effect.
Early clinical
An early human study for which a formal clinical phase is not assigned or publicly stated.
Biallelic
A disease-causing change is present in both copies of a gene.
Dual vector
Two delivery vehicles carry separate parts of a gene that is too large to fit into one vehicle.
Intracochlear
Delivered into the fluid-filled inner-ear structure involved in hearing.

Who is being studied

The population described by the public study record.

Study population
People aged 1 year and older with OTOF-related hearing loss
Age range
1 year and older
Planned enrollment
Not publicly disclosed
Study regions
China

This summary describes the study population; it cannot determine whether an individual qualifies.

Therapy design

The genetic treatment and how its activity is controlled.

Delivery vector and gene cargo
Dual Anc80L65 vectors encoding human OTOF, split between exons 20 and 21 with splice-donor/acceptor-mediated reconstitution
Promoter
mMyo15 hair-cell-specific promoter

Delivery to the inner ear

How the therapy is administered and whether one or both ears are treated.

How it is delivered
Intracochlear administration
How the inner ear is reached
Round-window membrane injection; preclinical program work used a transmastoid facial-recess corridor
Fenestration
No separate fenestration reported; round-window membrane injection
One or both ears
Cohort-dependent
Study start date
Jun 26, 2023

Public evidence reviewed

Evidence and limitations

Current through Jul 14, 2026

The NCT-linked human paper identifies the clinical vector capsid. Program-specific preclinical work resolves the dual-vector split, reconstitution strategy, mMyo15 promoter, and round-window route.

Details not publicly disclosed

  • Current registry status
  • Human operative corridor
Open the field-by-field evidence map →
Educational resource. This page summarizes public evidence and cannot determine whether someone qualifies for a study. Recruitment status, eligibility, and locations can change. Confirm current details with ClinicalTrials.gov and the study team, and discuss medical decisions with a qualified clinician.